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Temozolomide-modulated glioma proteome: Local bbw dating. Till date majority of proteomics studies in glioblastoma have focused on identification of differential proteins amongst different GBM cell lines, patient samples or within the same tumour to investigate the heterogeneity of glioblastoma, mechanism of chemoresistance and identification of diagnostic biomarkers [ 14 — 26 ].

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Proteasomes are well known targets in cancer therapy owing to their role in maintaining homeostasis of proteins involved in cell cycle, signalling pathways regulating cell survival and apoptosis [ 31 — 34 ].
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Luciferase values subsequent to normalization were plotted.






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Furthermore absence of proteasome activity in RR cells also significantly decreases their ability to form tumours in vivo.






Since the RR population exhibited increased proteasome activity we wanted to analyze if the survival of RR cells was dependent on the proteasome activity.



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Ann Neurol.






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NF-kappa B-mediated adaptive resistance to ionizing radiation.






Published online Jun



Deregulation of proteasome pathway in the radiation resistant population A Pathway analysis of deregulated genes in Radiation Resistant RR vs.






The ubiquitin-mediated protein degradation pathway in cancer:



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